Info Feature: Pet Obesity — New
Knowledge
Like obesity in humans (both adult and child),
obesity has become a serious health concern for
pets, to an epidemic level. Not only is it a concern
purely because of what we’ve known about it to this
point (obese pets live shorter lives, and are more
prone to heart and joint issues), but because of
what’s been discovered recently through new research
about the nature of obesity and its previously unknown
effects.
Let’s start with some causes. One highly contributing
factor is, obviously, a lack of activity. This is
more a predominant factor with dogs; cats have a
less active lifestyle in general and thus require
less activity. Dogs are designed by nature to move
around daily — a LOT. If they can’t get any
exercise, the pounds pile on. The other obvious
factor is the amount of food our pets are getting.
We sometimes tend to equate love with food, and
when we see our pets obviously enjoying their meal,
our minds, consciously or subconsciously, add just
a little more to the bowl. However, the pet food
manufacturers are also to blame. The feeding charts
on their bags tend to recommend too much. They’re
trying to sell more food, so it’s in their interest
to bump up the charts a little. However, the results
are often pudgy pals. The rich nutritional content
of pet foods, particularly the high-end foods, also
contributes. Dog and cat physiologies aren’t designed
to handle large quantities of very rich foods, so
if you’re not watching carefully, weight gain can
happen very quickly. The bottom line: don’t trust
feeding charts. Experiment with food levels and
keep visual track of your pet. (Don’t be concerned
with pounds.) There are two charts with this article
designed to help you do just that.
The newest research has found that obesity affects
pets at the cellular level. White fat does more
than store and release energy; it also secretes
cell-to-cell "communication" proteins. There’s a
ton of techno-babble that can be distilled into
a couple of fairly simple statements. First, obese
individuals don’t receive normal body signals that
help regulate appetite and energy usage. Those pets
are getting messages from their brains to eat too
much, because their brains aren’t telling them that
they’re full, and at the same time getting a message
to hold on to their fat, rather than use it for
energy. Another result of this cellular activity:
proteins are secreted by fat that increase resistance
to insulin (diabetes is therefore a common result
of chronic obesity), and increase inflammation.
Obesity is also a major contributor to osteoarthritis.
Simply put, being fat is hard on the joints. When
the damage to joints outpaces the body’s production
of healthy joint bone and tissue, osteoarthritis
develops.
Here’s another result: respiratory difficulty.
Obese pets, when in the midst of physical activity,
have breathing difficulties due to the constriction
of their airways and a shifting of their diaphragms
resulting from being displaced by so much fat. Obesity
can also contribute to a collapse of the trachea
(usually seen with exertion coupled with a cough).
concluded on page 3

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